The strong, malty black tea from India. Black tea helps your blood vessels relax better, and regular drinkers live somewhat longer.
Mechanisms, studies and evidence grades for each claim above.
Assam is black tea from the Brahmaputra valley in northeast India, made from the large-leaf Camellia sinensis var. assamica (the same variety pu-erh is made from). Black tea is fully oxidized: leaf enzymes turn most of the green-tea catechins (EGCG and others) into theaflavins (orange-red dimers, ~2–6% of dry solids) and thearubigins (larger, heterogeneous brown polymers, the bulk of the pigment). Assam's high polyphenol content and strong, malty taste are why it's the base of most "breakfast" blends and of Indian chai. Assam itself has no trial literature. The evidence below is for black tea, and Assam is a strong example of it.
In a crossover RCT in 66 patients with coronary artery disease, black tea improved endothelium-dependent flow-mediated dilation (FMD) of the brachial artery both 2 hours after 450 mL and after 4 weeks of 900 mL/day. Water had no effect. An equal dose of caffeine alone (200 mg) did nothing, so the effect comes from the flavonoids, not the caffeine. Nitroglycerin-induced dilation didn't change either, which means the improvement is in the endothelium's own nitric-oxide production rather than in the smooth muscle.1 Cell work points the same way: tea flavonoids activate endothelial NO synthase by phosphorylating it.2 (Moderate — several small RCTs with consistent FMD results)
This is the same endpoint — more NO, better arterial relaxation — that beets reach through nitrate. It's another route into the pathway: tea activates eNOS directly, beets supply nitrate that becomes NO without eNOS. A 6-month RCT of 3 cups a day also found a small drop in blood pressure,3 but the change in vessel function is the more meaningful finding. Note that the blood-pressure trial's co-authors included Unilever (Lipton) staff.
In 16 healthy women, 500 mL black tea improved FMD, and the same tea with 10% skimmed milk had no effect. The authors traced this to caseins binding tea catechins.2 The UK Biobank cohort below, whose members mostly drink their tea with milk, still showed lower cardiovascular mortality. So milk may blunt the acute effect without cancelling the long-term association. If vessel function is the reason you're drinking it, drink it black.
↑ back to summaryAn NCI analysis of about 500,000 UK Biobank participants, most of them black-tea drinkers, followed for a median of 11.2 years found lower all-cause mortality from 2 cups a day upward. Hazard ratios were about 0.87–0.88 for 2–7 cups a day and stayed around 0.89 even at 10+ cups. The association held for cardiovascular disease, ischemic heart disease and stroke, whether or not people also drank coffee, and regardless of genetic caffeine-metabolism speed.4 (Moderate — large, well-adjusted cohort, but still observational; tea strength and portion size weren't measured)
↑ back to summaryA cup of Assam has roughly 40–70 mg caffeine (about half a coffee) plus some L-theanine. The trials of the combination are covered on the theanine page. A cup of tea has far less theanine than those trial doses, but the direction is the same.
↑ back to summaryOxalate and fluoride: black tea is a meaningful oxalate source for people who form kidney stones, and in very heavy drinkers (a liter or more a day for years) fluoride can build up, as with pu-erh bricks. Caffeine: cut back in the afternoon if sleep is fragile.
↑ back to summaryThe five references were checked against PubMed in this session (Hodgson 2012 is a research letter with no abstract; its design comes from the published letter, and its authors include Unilever staff). The theaflavin percentage, the per-cup caffeine range and the oxalate/fluoride notes have no numbered citation yet.